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mouse anti ck19  (Developmental Studies Hybridoma Bank)


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    Structured Review

    Developmental Studies Hybridoma Bank mouse anti ck19
    (A) Disease burden (% disease/total pancreas area) for male KC mice fed control diet (CD) or designated HFDs for 12 weeks. Box plots designate 25 th , 50 th , and 75 th percentiles +/- min/max, n = 4-12 mice/diet. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (B) Positive correlation between median disease burden of mice in (A) and dietary oleic acid concentration (µg/mg diet). Pearson correlation coefficient (R) and p -value are shown. (C) Stronger positive correlation between median disease burden of mice in (A) and mean total plasma oleic acid levels (nmol of FA/mL plasma) in non-tumor-bearing C57BL/6 male mice fed each diet for 12 weeks (n = 5 mice/diet). Pearson correlation coefficient (R) and p -value are shown. (D) Representative H&E and <t>Ck19</t> (duct tumor marker) immunostaining of pancreata of male KC mice fed CD, LODs, or HODs for 12 weeks. Scale bars are 50 µm. (E) Disease burden (mean +/- s.e.m., n = 7-12 mice/diet) for male KC mice fed CD, LODs, or HODs for 12 weeks. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (F) Proportional area (mean percentage +/- s.e.m., n = 6-9 mice/diet) of total disease by lesion type (ADM, PanIN, PDAC) of KC mice fed CD, LODs, or HODs. Proportions are not significantly different across diets, Kruskal-Wallis test with Dunn’s post-hoc test.
    Mouse Anti Ck19, supplied by Developmental Studies Hybridoma Bank, used in various techniques. Bioz Stars score: 97/100, based on 253 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+ck19/bio_rxiv__2025__04__04__645324-249-6-8?v=Developmental+Studies+Hybridoma+Bank
    Average 97 stars, based on 253 article reviews
    mouse anti ck19 - by Bioz Stars, 2026-07
    97/100 stars

    Images

    1) Product Images from "Diet-induced phospholipid remodeling dictates ferroptosis sensitivity and tumorigenesis in the pancreas"

    Article Title: Diet-induced phospholipid remodeling dictates ferroptosis sensitivity and tumorigenesis in the pancreas

    Journal: bioRxiv

    doi: 10.1101/2025.04.04.645324

    (A) Disease burden (% disease/total pancreas area) for male KC mice fed control diet (CD) or designated HFDs for 12 weeks. Box plots designate 25 th , 50 th , and 75 th percentiles +/- min/max, n = 4-12 mice/diet. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (B) Positive correlation between median disease burden of mice in (A) and dietary oleic acid concentration (µg/mg diet). Pearson correlation coefficient (R) and p -value are shown. (C) Stronger positive correlation between median disease burden of mice in (A) and mean total plasma oleic acid levels (nmol of FA/mL plasma) in non-tumor-bearing C57BL/6 male mice fed each diet for 12 weeks (n = 5 mice/diet). Pearson correlation coefficient (R) and p -value are shown. (D) Representative H&E and Ck19 (duct tumor marker) immunostaining of pancreata of male KC mice fed CD, LODs, or HODs for 12 weeks. Scale bars are 50 µm. (E) Disease burden (mean +/- s.e.m., n = 7-12 mice/diet) for male KC mice fed CD, LODs, or HODs for 12 weeks. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (F) Proportional area (mean percentage +/- s.e.m., n = 6-9 mice/diet) of total disease by lesion type (ADM, PanIN, PDAC) of KC mice fed CD, LODs, or HODs. Proportions are not significantly different across diets, Kruskal-Wallis test with Dunn’s post-hoc test.
    Figure Legend Snippet: (A) Disease burden (% disease/total pancreas area) for male KC mice fed control diet (CD) or designated HFDs for 12 weeks. Box plots designate 25 th , 50 th , and 75 th percentiles +/- min/max, n = 4-12 mice/diet. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (B) Positive correlation between median disease burden of mice in (A) and dietary oleic acid concentration (µg/mg diet). Pearson correlation coefficient (R) and p -value are shown. (C) Stronger positive correlation between median disease burden of mice in (A) and mean total plasma oleic acid levels (nmol of FA/mL plasma) in non-tumor-bearing C57BL/6 male mice fed each diet for 12 weeks (n = 5 mice/diet). Pearson correlation coefficient (R) and p -value are shown. (D) Representative H&E and Ck19 (duct tumor marker) immunostaining of pancreata of male KC mice fed CD, LODs, or HODs for 12 weeks. Scale bars are 50 µm. (E) Disease burden (mean +/- s.e.m., n = 7-12 mice/diet) for male KC mice fed CD, LODs, or HODs for 12 weeks. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (F) Proportional area (mean percentage +/- s.e.m., n = 6-9 mice/diet) of total disease by lesion type (ADM, PanIN, PDAC) of KC mice fed CD, LODs, or HODs. Proportions are not significantly different across diets, Kruskal-Wallis test with Dunn’s post-hoc test.

    Techniques Used: Control, Concentration Assay, Marker, Immunostaining



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    Developmental Studies Hybridoma Bank mouse anti ck19
    (A) Disease burden (% disease/total pancreas area) for male KC mice fed control diet (CD) or designated HFDs for 12 weeks. Box plots designate 25 th , 50 th , and 75 th percentiles +/- min/max, n = 4-12 mice/diet. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (B) Positive correlation between median disease burden of mice in (A) and dietary oleic acid concentration (µg/mg diet). Pearson correlation coefficient (R) and p -value are shown. (C) Stronger positive correlation between median disease burden of mice in (A) and mean total plasma oleic acid levels (nmol of FA/mL plasma) in non-tumor-bearing C57BL/6 male mice fed each diet for 12 weeks (n = 5 mice/diet). Pearson correlation coefficient (R) and p -value are shown. (D) Representative H&E and <t>Ck19</t> (duct tumor marker) immunostaining of pancreata of male KC mice fed CD, LODs, or HODs for 12 weeks. Scale bars are 50 µm. (E) Disease burden (mean +/- s.e.m., n = 7-12 mice/diet) for male KC mice fed CD, LODs, or HODs for 12 weeks. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (F) Proportional area (mean percentage +/- s.e.m., n = 6-9 mice/diet) of total disease by lesion type (ADM, PanIN, PDAC) of KC mice fed CD, LODs, or HODs. Proportions are not significantly different across diets, Kruskal-Wallis test with Dunn’s post-hoc test.
    Mouse Anti Ck19, supplied by Developmental Studies Hybridoma Bank, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+ck19/bio_rxiv__2025__04__04__645324-249-6-8?v=Developmental+Studies+Hybridoma+Bank
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    Image Search Results


    (A) Disease burden (% disease/total pancreas area) for male KC mice fed control diet (CD) or designated HFDs for 12 weeks. Box plots designate 25 th , 50 th , and 75 th percentiles +/- min/max, n = 4-12 mice/diet. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (B) Positive correlation between median disease burden of mice in (A) and dietary oleic acid concentration (µg/mg diet). Pearson correlation coefficient (R) and p -value are shown. (C) Stronger positive correlation between median disease burden of mice in (A) and mean total plasma oleic acid levels (nmol of FA/mL plasma) in non-tumor-bearing C57BL/6 male mice fed each diet for 12 weeks (n = 5 mice/diet). Pearson correlation coefficient (R) and p -value are shown. (D) Representative H&E and Ck19 (duct tumor marker) immunostaining of pancreata of male KC mice fed CD, LODs, or HODs for 12 weeks. Scale bars are 50 µm. (E) Disease burden (mean +/- s.e.m., n = 7-12 mice/diet) for male KC mice fed CD, LODs, or HODs for 12 weeks. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (F) Proportional area (mean percentage +/- s.e.m., n = 6-9 mice/diet) of total disease by lesion type (ADM, PanIN, PDAC) of KC mice fed CD, LODs, or HODs. Proportions are not significantly different across diets, Kruskal-Wallis test with Dunn’s post-hoc test.

    Journal: bioRxiv

    Article Title: Diet-induced phospholipid remodeling dictates ferroptosis sensitivity and tumorigenesis in the pancreas

    doi: 10.1101/2025.04.04.645324

    Figure Lengend Snippet: (A) Disease burden (% disease/total pancreas area) for male KC mice fed control diet (CD) or designated HFDs for 12 weeks. Box plots designate 25 th , 50 th , and 75 th percentiles +/- min/max, n = 4-12 mice/diet. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (B) Positive correlation between median disease burden of mice in (A) and dietary oleic acid concentration (µg/mg diet). Pearson correlation coefficient (R) and p -value are shown. (C) Stronger positive correlation between median disease burden of mice in (A) and mean total plasma oleic acid levels (nmol of FA/mL plasma) in non-tumor-bearing C57BL/6 male mice fed each diet for 12 weeks (n = 5 mice/diet). Pearson correlation coefficient (R) and p -value are shown. (D) Representative H&E and Ck19 (duct tumor marker) immunostaining of pancreata of male KC mice fed CD, LODs, or HODs for 12 weeks. Scale bars are 50 µm. (E) Disease burden (mean +/- s.e.m., n = 7-12 mice/diet) for male KC mice fed CD, LODs, or HODs for 12 weeks. * p < 0.05, ** p < 0.01, Kruskal-Wallis test with Dunn’s post-hoc test. (F) Proportional area (mean percentage +/- s.e.m., n = 6-9 mice/diet) of total disease by lesion type (ADM, PanIN, PDAC) of KC mice fed CD, LODs, or HODs. Proportions are not significantly different across diets, Kruskal-Wallis test with Dunn’s post-hoc test.

    Article Snippet: The following primary antibodies were used: mouse anti-Ck19 (DSHB TROMA-III, 1:500), rabbit anti-Ki67 (Biocare Medical CRM325, 1:200), rat anti-p19 ARF (Santa Cruz Biotech sc-32748, 1:100), rat anti-Cd45 (Abcam ab25386, 1:100), rabbit anti-F4/80 (CST 70076, 1:400), rabbit anti-SMA (Invitrogen PA1-37024, 1:100), mouse anti-B220 (BD Pharmingen 550286, 1:40), rabbit anti-Cd3 (CST 78588, 1:200), rabbit anti-4-HNE (Abcam, ab46545, 1:200), and rabbit anti-Nqo1 (Sigma-Aldrich HPA007308, 1:100).

    Techniques: Control, Concentration Assay, Marker, Immunostaining